Preliminary experiments reveal conolidine might inhibit distinct ion channels, decreasing neuronal excitability and restricting agony indicators. This system is especially related in neuropathic pain, where abnormal signaling results in persistent soreness. Furthermore, conolidine appears to affect G protein-coupled receptor (GPCR) pathways integral to ache perception. Computational d... https://scotty061tlb7.wikicarrier.com/user